Pin1 promotes human Ca V 2.1 channel polyubiquitination by RNF138: pathophysiological implication for episodic ataxia type 2.
Fu SJ; Cheng KM; Hsiao CT; Fang YC; Jeng CJ; Tang CY · 2024 · Cell communication and signaling : CCS
WASTE classifies this as Negative / Null Result Report · AI classification, approximate
The study found no significant effect — useful as a negative control or null benchmark for your own design.
Abstract (excerpt)
Loss-of-function mutations in the human gene encoding the neuron-specific Ca 2+ channel Ca V 2.1 are linked to the neurological disease episodic ataxia type 2 (EA2), as well as neurodevelopmental disorders such as developmental delay and…
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Metadata source: Europe PMC · DOI 10.1186/s12964-024-01960-9